烧伤学临床新视野:烧伤休克、感染、营养、修复与整复(第2版)
上QQ阅读APP看书,第一时间看更新

参考文献

董宁,姚咏明,曹玉珏,等,2007.严重烧伤患者人白细胞抗原DR定量表达的临床意义[J] .中华外科杂志,45:766—769.

脓毒症免疫调理治疗临床研究协作组,2007.乌司他丁、α1胸腺肽联合治疗严重脓毒症——一种新的免疫调理治疗方法的临床研究[J] .中华医学杂志,87:451—457.

盛志勇,姚咏明,2011.加强对脓毒症免疫功能障碍及其监测的研究[J] .解放军医学杂志,36:8—10.

盛志勇,姚咏明,林洪远,等,2002.全身炎症反应和多器官功能障碍综合征认识的变迁及形状[J] .解放军医学杂志,27:98—100.

王正国,2003.脓毒症研究概况[J] .中华创伤杂志,19:5—8.

姚咏明,2012.重视脓毒症免疫功能的负向调控作用[J] .中华急诊医学杂志,21:117—119.

姚咏明,柴家科,盛志勇,等,2003.烧伤脓毒症的诊断标准与防治[J] .中华烧伤杂志,19:65—66.

姚咏明,黄立锋,2011.烧伤后脓毒性脑病发生机制与诊治对策[J] .中华损伤与修复杂志,6:167—173.

姚咏明,黄立锋,2011.调节性T淋巴细胞与严重烧伤脓毒症[J] .中华烧伤杂志,27:81—83.

姚咏明,盛志勇,2003.我国创伤脓毒症基础研究新进展[J] .中华创伤杂志,19:9—12.

姚咏明,盛志勇,2004.金黄色葡萄球菌外毒素与脓毒症及多器官损害[J] .中华创伤杂志,20:711—714.

姚咏明,盛志勇,柴家科,等,2008.烧伤脓毒症发病机制与防治对策[J] .中华烧伤杂志,24:337—339.

姚咏明,盛志勇,林洪远,等,2004.脓毒症定义及诊断的新认识[J] .中国危重病急救医学,16:321—324.

张庆红,姚咏明,2010.关注神经内分泌紊乱与脓毒症的关系及其防治策略[J] .中华烧伤杂志, 26:87—89.

张庆红,姚咏明,2011.浅析脓毒症高血糖治疗的利与弊[J] .中华急诊医学杂志, 20:789—791.

ANGUS D C, VAN DER POLL T, 2013. Severe sepsis and septic shock [J]. N Engl J Med, 369:840—851.

ANNANE D, BELLISSANT E, BOLLAERT P E, et al, 2009. Corticosteroids in the treatment of severe sepsis and septic shock in adults: a systematic review [J]. JAMA, 301: 2362—2375.

BERCZI I, QUINTANAR-STEPHANO A, KOVACS K, et al, 2009. Neuroimmune regulation in immunocompetence, acute illness, and healing [J]. Ann N Y Acad Sci, 1153: 220—239.

CHENG B L, XIE G H, YAO S L, et al, 2007. Epidemiology of severe sepsis in critically ill surgical patients in ten university hospitals in China [J]. Crit Care Med, 35: 2538—2546.

DELLINGER P R, LEVY M M, RHODES A, et al, 2013. Surviving sepsis campaign: international guidelines for management of severe sepsis and septic shock 2012[J]. Crit Care Med, 41: 580—637.

HATTORI Y, TAKANO K, TERAMAE H, et al, 2010. Insights into sepsis therapeutic design based on the apoptotic death pathway [J]. J Pharmacol Sci, 114: 354—365.

HOTCHKISS R S, OPAL S, 2010. Immunotherapy for sepsis: a new approach against an ancient foe [J]. N Engl J Med, 363: 87—89.

HUANG L F, YAO Y M, DONG N, et al, 2010. Association between regulatory T cell activity and sepsis and outcome of severely burned patients:a prospective, observational study [J]. Crit Care, 14: R3.

HUTTUNEN R, AITTONIEMI J, 2011. New concepts in the pathogenesis, diagnosis and treatment of bacteremia and sepsis [J]. J Infect, 63:407—419.

JIANG L N, YAO Y M, SHENG Z Y, et al, 2012. The role of regulatory T cells in the pathogenesis of sepsis and its clinical implication [J]. J Interferon Cytokine Res, 32: 341—349.

LI H Y, YAO Y M, SHI Z G, et al, 2003. The potential role of Staphylococcal enterotoxin B in rats with postburn Staphylococcus aureus sepsis [J] . Shock, 20: 257—263.

LIN J, YAO Y M, DONG N, et al, 2009. Influence of CD14 polymorphism on CD14 expression in patients with extensive burns [J]. Burns, 35:365—371.

LIN M T, ALBERTSON T E, 2004. Genomic polymorphisms in sepsis [J]. Crit Care Med, 32: 569—579.

LIU H, YAO Y M, WANG S B, et al, 2009. Inhibition of Janus kinase 2 and signal transduction and activator of transcription 3 protect against cecal ligation and puncture-induced multiple organ damage and mortality [J]. J Trauma, 66: 859—865.

LUAN Y Y, YAO Y M, SHENG Z Y, et al, 2012. Update on the immunological pathway of negative regulation in acute insults and sepsis [J]. J Interferon Cytokine Res, 32: 288—298.

RUSSELL J A, 2006. Management of sepsis [J]. N Engl J Med, 355: 1699—1713.

TERRAGNI P P, ROSBOCH G, TEALDI A, et al, 2007. Tidal hyperinflation during low tidal volume ventilation in acute respiratory distress syndrome [J]. Am J Respir Crit Care Med, 175: 160—166.

WIENER R S, WIENER D C, LARSON R J, et al, 2008. Benefits and risks of tight glucose control in critically ill adults: a meta-analysis [J] . JAMA, 300: 933—944.

YAO Y M, SHENG Z Y, HUANG L F, et al, 2009. The effect of a novel cytokine, high mobility group box-1 protein, on the development of traumatic sepsis [J]. Chin J Integr Med, 15: 13—15.

ZHANG L T, YAO Y M, LU J Q, et al, 2008. Recombinant bactericidal/permeability- increasing protein inhibits endotoxin-induced high mobility group box 1 protein gene expression in sepsis [J]. Shock, 29: 278—284.

ZHANG Q H, YAO Y M, SHENG Z Y, et al, 2009. Dysfunction of neuroendocrine system in sepsis and implication of hormone therapy [J]. J Geriatr Cardiol, 6: 249—254.

ZHU X M, YAO F H, YAO Y M, et al, 2012. Endoplasmic reticulum stress and its regulator XBP-1 contributes to dendritic cell maturation and activation induced by high mobility group box-1 protein [J]. Int J Biochem Cell Biol, 44: 1097—1105.