参考文献
陈欣,张雅莲,孙永华,等,1997.我院耐甲氧西林金黄色葡萄球菌的耐药性及肠毒素分析[J] .中华整形烧伤外科杂志,13:377—379.
方文慧,姚咏明,施志国,等,1999.杀菌/通透性增加蛋白对烫伤大鼠脂多糖结合蛋白和CD14 mRNA表达的影响[J] .中华医学杂志,79:289—291.
方文慧,姚咏明,施志国,等,1999.烫伤大鼠内毒素的组织分布特点及意义[J] .中华整形烧伤外科杂志,15:298—300.
方文慧,姚咏明,施志国,等,1999.烫伤后脂多糖受体CD14和肿瘤坏死因子-α基因表达的变化规律[J] .中华外科杂志,37:271—273.
李红云,姚咏明,施志国,等,2000.金葡菌肠毒素B单抗对烫伤脓毒症大鼠脏器功能的影响[J] .中华医学杂志,80:872—874.
李红云,姚咏明,施志国,等,2001.金黄色葡萄球菌肠毒素B单克隆抗体对烫伤脓毒症大鼠急性肺损伤的影响[J] .中华实验外科杂志,18:228—230.
李红云,姚咏明,施志国,等,2002.双单抗夹心ELISA方法检测血浆及组织金黄色葡萄球菌肠毒素B[J] .中华医院感染学杂志,12:655—657.
李红云,姚咏明,姚凤华,等,2004.烫伤脓毒症大鼠细胞因子信号转导抑制因子基因表达的改变及其意义[J] .中华创伤杂志,20:734—738.
施志国,张延霞,刘伟,等,1994.金黄色葡萄球菌肠毒素和中毒性休克毒素的分析[J] .中华医院感染学杂志,4:69—71.
杨丽萍,姚咏明,李杰萍,等,2009.STAT3与MAPK蛋白协同调节肿瘤坏死因子-α转录活性[J] .生物化学与生物物理进展,36:1003—1011.
杨丽萍,姚咏明,叶棋浓,等,2011.信号转导和转录激活因子3调节肿瘤坏死因子-α表达的结合位点研究[J] .生物化学与生物物理进展,38:1145—1152.
姚咏明,2005.内毒素与革兰阳性菌致病因子的协同效应与意义[J] .中国危重病急救医学,17:193—196.
姚咏明,2009.创伤感染并发症免疫功能障碍及其诊治的若干问题[J] .中华外科杂志,47:37—39.
姚咏明,柴家科,林洪远,等,2005.现代脓毒症理论与实践[M] .北京:科学出版社,96—145.
姚咏明,程明华,姚胜,等,2004.细胞外信号调节激酶抑制剂对烫伤脓毒症大鼠肿瘤坏死因子-α表达的影响及意义[J] .中华外科杂志,42:391—395.
姚咏明,栾樱译,2011.提高对创伤感染及其并发症的认识[J] .临床急诊杂志,12:361—364.
姚咏明,盛志勇,2003.我国创伤脓毒症基础研究新进展[J] .中华创伤杂志,19:9—12 .
姚咏明,盛志勇,2004.金黄色葡萄球菌外毒素与脓毒症及多器官损害[J] .中华创伤杂志,20:711—714.
姚咏明,盛志勇,2005.加强对烧伤后金黄色葡萄球菌外毒素作用的研究[J] .中华烧伤杂志,21:152—154.
姚咏明,盛志勇,2005.重视对脓毒症本质的探讨[J] .中华急诊医学杂志,14:185—186.
姚咏明,田惠民,王亚平,等,1992.Re型内毒素抗血清对实验性多器官功能衰竭的防护作用[J] .中华外科杂志,30:244—247.
姚咏明,王亚平,田惠民,等,1993.严重烧伤内毒素血症的临床意义[J] .中华外科杂志,31:435—438.
姚咏明,于燕,吴叶,等,1998.烧伤后多器官功能障碍综合征患者可溶性脂多糖受体CD14的变化[J] .中华外科杂志,36:668—670.
姚咏明,于燕,盛志勇,等,1999.肠缺血-再灌注损伤后内毒素增敏作用及其机制的初步探讨[J] .中华整形烧伤外科杂志,15:301—304.
张庆红,姚咏明,2010.浅析外科感染的免疫障碍问题[J] .中国普外基础与临床杂志,17:1129—1131.
AZUMA K, KOIKE K, KOBAYASHI T, et al, 2004. Detection of circulating superantigens in an intensive care unit population [J]. Int J Infect Dis, 8: 92—98.
BIN L, ZHANG R, LI J, et al, 2008. Antimalarial artesunate protects sepsis model mice against heat-killed Escherichia coli challenge by decreasing TLR4, TLR9 mRNA expressions and transcription factor NF-κB activation[J]. Int Immunopharmacol, 8: 379—389.
BLANK C, LUZ A, BENDIGS S, et al, 1997. Superantigen and endotoxin synergize in the induction of lethal shock[J]. Eur J Immunol, 27:825—833.
COHEN J, 2000. The dectection and interpretation of endotoxaemia[J]. Inten Care Med, 26: S51—S56.
DONG Y L, KO F, YAN T, et al, 1993. Evidence for Kupffer cell activation by burn injury and pseudomonas exotoxin A[J]. Burns, 19: 12—16.
FANG W H, YAO Y M, SHI Z G, et al, 2001. Effect of recombinant bactericidal/ permeability-increasing protein on endotoxin translocation and lipopolysaccharide- binding protein/CD14 expression in rats following thermal injury[J]. Crit Care Med, 29: 1452—1459.
FANG W H, YAO Y M, SHI Z G, et al, 2002. Lipopolysaccharide-binding protein and lipopolysaccharide receptor CD14 gene expression after thermal injury and its potential mechanism(s)[J]. J Trauma, 53: 957—967.
FANG W H, YAO Y M, ZHAI H X, et al, 2004. Tissue lipopolysaccharide-binding protein expression in rats after thermal injury: potential role of TNF-α[J]. Burns, 30: 225—231.
FAURE E, THOMAS L, XU H, et al, 2001. Bacterial lipopolysaccharide and IFN-gamma induce Toll-like receptor 2 and Toll-like receptor 4 expression in human endothelial cells: role of NF-kappa B activation[J]. J Immunol, 166: 2018—2024.
GAESTEL M, KOTLYAROV A, KRACHT M, et al, 2009. Targeting innate immunity protein kinase signalling in inflammation[J]. Nat Rev Drug Discov, 8: 480—499.
JACOBS A T, IGNARRO L J, 2001. Lipopolysaccharide-induced expression of interferon-beta mediates the timing of inducible nitric-oxide synthase induction in RAW 2647 macrophages[J]. J Biol Chem, 276: 47950—47957.
JUN W, ZHOU H, ZHENG J, et al, 2006. Antimalarial artemisinin synergizes with antibiotics to protect against lethal live Escherichia coli by decreasing proinflammatory cytokine release[J]. Antimicrob Agents Chemother, 50: 2420—2427.
KABELITZ D, 2007. Expression and function of Toll-like receptors in T lymphocytes[J]. Curr Opin Immunol, 19: 39—45.
KLEIN D J, BRIET F, NISENBAUM R, et al, 2011. Endotoxemia related to cardiopulmonary bypass is associated with increased risk of infection after cardiac surgery: a prospective observational study[J]. Crit Care, 15: R69.
KUMAR A, BUNNELL E, LYNN M, et al, 2004. Experimental human endotoxemia is associated with depression of load-independent contractility indices: prevention by the lipid a analogue E5531[J]. Chest, 126: 860—867.
LEEMANS J C, HEIKENS M, VAN KESSEL K P, et al, 2003. Lipoteichoic acid and peptidoglycan from Staphylococcus aureus synergistically induce neutrophil influx into the lungs of mice[J]. Clin Diagn Lab Immunol, 10: 950—953.
LI H Y, YAO Y M, SHI Z G, et al, 2003. Significance of biopterin induction in rats with postburn Staphylococcus aureus sepsis[J]. Shock, 20:159—165.
LI H Y, YAO Y M, SHI Z G, et al, 2003. The potential role of Staphylococcal enterotoxin B in rats with postburn Staphylococcus aureus sepsis [J] . Shock, 20: 257—263.
LIN J, YAO Y M, DONG N, et al, 2009. Influence of CD14 polymorphism on CD14 expression in patients with extensive burns[J]. Burns, 35:365—371.
LIU H, YAO Y M, YU Y, et al, 2007. Role of Janus kinase/signal transducer and activator of transcription pathway in regulation of expression and inflammation-promoting activity of high mobility group box protein 1 in rat peritoneal macrophages[J]. Shock, 27: 55—60.
MARSHALL J C, FOSTER D, VINCENT J L, et al, 2004. Diagnostic and prognostic implications of endotoxemia in critical illness: results of the MEDIC study[J]. J Infect Dis, 190: 527—534.
MARTIN G S, MANNINO D M, EATON S, et al, 2003. The epidemiology of sepsis in the United States from 1979 through 2000[J]. N Engl J Med, 348: 1546—1554.
MITAKA C, TOMITA M, 2011. Polymyxin B-immobilized fiber column hemoperfusion therapy for septic shock[J]. Shock, 36: 332—338.
OPAL S M, COHEN J, 1999. Clinical gram-positive sepsis: does it fundamentally differ from gram-negative bacterial sepsis?[J]. Crit Care Med, 27: 1608—1616.
OTTE J M, CARIO E, PODOLSKY D K, et al, 2004. Mechanisms of cross hyporesponsiveness to toll-like receptor bacterial ligands in intestinal epithelial cells[J]. Gastroenterology, 126: 1054—1070.
PULEO F, ARVANITAKIS M, VAN GOSSUM A, et al, 2011. Gut failure in the ICU[J]. Semin Respir Crit Care Med, 32: 626—638.
REDL H, BAHRAMI S, SCHLAG G, et al, 1993. Clinical detection of LPS and animal models of endotoxemia[J]. Immunobiology, 187: 330—345.
RIJNDERS M I, DEURENBERG R H, BOUMANS M L, et al, 2009. Population structure of Staphylococcus aureus strains isolated from intensive care unit patients in the netherlands over an 11-year period (1996 to 2006)[J]. J Clin Microbiol, 47: 4090—4095.
SAIDO S H, ISHIBASHI J, MOMOTANI E, et al, 2004. In vitro and in vivo activity of antimicrobial peptides synthesized based on the insect defensin[J]. Peptides, 25: 19—27.
SCHLIEVERT P M, 1993. Role of superantigens in human disease[J]. J Infect Dis, 167: 997—1002.
SHIRANEE S, COHEN J, 1999. Gram-positive sepsis: mechanisms and differences from gram-negative sepsis [J]. Infect Dis Clin Nor Am, 13:397—412.
SHUAI K, 1999. The STAT family of proteins in cytokine signaling[J]. Pro Biophys Mol Biol, 71: 405—420.
STEPHENS R C, FIDLER K, WILSON P, et al, 2006. Endotoxin immunity and the development of the systemic inflammatory response syndrome in critically ill children[J]. Intensive Care Med, 32: 286—294.
SULIBURK J, HELMER K, MOORE F, et al, 2008. The gut in systemic inflammatory response syndrome and sepsis: Enzyme systems fighting multiple organ failure[J]. Eur Surg Res, 40: 184—189.
UCHIYAMA T, YAN X J, IMANISHI K, et al, 1994. Bacterial superantigens-mechanism of T cell activation by the superantigens and their role in the pathogenesis of infectious diseases[J]. Microbiol Immunol, 38: 245—256.
UKLEJA A, 2010. Altered GI motility in critically ill patients: current understanding of pathophysiology, clinical impact, and diagnostic approach
[J]. Nutr Clin Pract, 25: 16—25.
VENET C, ZENI F, VIALLON A, et al, 2000. Endotoxaemia in patients with severe sepsis or septic shock[J]. Intensive Care Med, 26: 538—544.
WANG J E, DAHLE M K, YNDESTAD A, et al, 2004. Peptidoglycan of Staphylococcus aureus causes inflammation and organ injury in the rat [J] .Crit Care Med, 32: 546—552.
WIERSINGA W J, 2011. Current insights in sepsis: from pathogenesis to new treatment targets[J]. Curr Opin Crit Care, 17: 480—486 .
YAGUCHI A, YUZAWA J, KLEIN D J, et al, 2012. Combining intermediate levels of the Endotoxin Activity Assay (EAA) with other biomarkers in the assessment of patients with sepsis: results of an observational study[J]. Crit Care, 16: R88.
YAO Y M, BAHRAMI S, LEICHTFRIED G, et al, 1995. Pathogenesis of hemorrhage-induced bacteria/ endotoxin translocation in rats: effects of recombinant bactericidal/ permeability-increasing protein[J]. Ann Surg, 221: 398—405.
YAO Y M, BAHRAMI S, REDL H, et al, 1996. Monoclonal antibody to tumor necrosis factor—a attenuates hemodynamic dysfunction secondary to intestinal ischemia/ reperfusion in rats[J]. Crit Care Med, 24: 1547—1553.
YAO Y M, LU L R, YU Y, et al, 1997. The influence of selective decontamination of the digestive tract on cell-mediated immune function and bacteria/endotoxin translocation in thermally injured rats[J]. J Trauma, 42: 1073—1079.
YAO Y M, REDL H, BAHRAMI S, et al, 1998. The inflammatory basis of trauma/shock associated multiple organ failure[J]. Inflamm Res, 47:74. 201—210.
YAO Y M, SHENG Z Y, TIAN H M, et al, 1995. The association of circulating endotoxaemia with the development of multiple organ failure in burned patients[J]. Burns, 21: 255—258.
YAO Y M, SHENG Z Y, YU Y, et al, 1995. The potential etiologic role of tumor necrosis factor in mediating multiple organ dysfunction in rats following intestinal ischemia-reperfusion injury[J]. Resuscitation, 29: 157—168.
YAO Y M, TIAN H M, SHENG Z Y, et al, 1995. Inhibitory effects of low-dose polymyxin B on hemorrhage-induced endotoxin/bacterial translocation and cytokine formation[J]. J Trauma, 38: 924—930.
YAO Y M, WANG Y P, TIAN H M, et al, 1996. Reduction of circulating prostaglandin E2 level by antiserum against core lipopolysaccharide in a rabbit model of multiple organ failure[J]. J Trauma, 40: 270—277.
ZHANG L T, YAO Y M, LU J Q, et al, 2008. Recombinant bactericidal/ permeability- increasing protein inhibits endotoxin-induced high mobility group box 1 protein gene expression in sepsis[J]. Shock, 29: 278—284.