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参考文献
1. Charles NS,John S.Resolution of inflammation:the beginning programs the end.Nature Immunology,2005,6(12):1191-1197.
2. Angus DC,van der Poll,T.Severe Sepsis and Septic Shock.N Engl J Med,2013,369(9):840-851.
3. Melamed A,Sorvillo FJ.The burden of sepsis-associated mortality in the United States from 1999 to 2005:an analysis of multiple-cause-of-death data.Crit Care,2009,13:R28.
4. Lagu T,Rothberg MB,Shieh MS,et al. Hospitalizations,costs,and outcomes of severe sepsis in the United States 2003 to 2007.Crit Care Med,2012,40:754-756.[Erratum,Crit Care Med 2012,40:2932.]
5. Das UN.Can sepsis and other critical illnesses be predicted and prognosticated?Adv Sepsis,2006,5:52-59.
6. Kinasewitz GT,Yan BS,Basson B,et al.For the PROWESS Sepsis Study Group.Universal changes in biomarkers of coagulation and inflammation occur in patients with severe sepsis,regardless of causative micro-organism[ISRCTN74215569]. Crit Care,2004,8:R82-90.
7. Karlsson S,Pettilä V,Tenhunen J,et al.HMGB1 as a predictor of organ dysfunction and outcome in patients with severe sepsis.Intensive Care Med,2008,34:1046-1053.
8. Yang H,Ochani M,Li J,et al.Reversing established sepsis with antagonists of endogenous high mobility group box-1. Proc Natl Acad Sci USA,2004,101:296-301.
9. Sunden-Cullberg J,Norrby-Teglund A,Rouhiainen A,et al. Persistent elevation of high mobility group box-1 protein (HMGB1)in patients with severe sepsis and septic shock. Crit Care Med,2005,33:564-573.
10. Felmet KA,Hall MW,Clark RS,et al.Prolonged lymphopenia,lymphoid depletion,and hypoprolactinemia in children with nosocomial sepsis and multiple organ failure.J Immunol,2005,174:3765-3772.
11. Arbour NC,Lorenz E,Schutte BC,et al.TLR4 mutations are associated with endotoxin hyporesponsiveness in humans. Nat Genet,2000,25:187-191.
12. Das UN.Critical advances in septicemia and septic shock. Crit Care,2000,4:290-294.
13. Shishido T,Nozaki N,Takahashi H,et al.Central role of endogenous toll-like receptor-2 activation in regulating inflammation,reactive oxygen species production,and subsequent neointimal formation after vascular injury.Biochem Biophys Res Commun,2006,345:1446-1453.
14. Norris PC,Reichart D,Dumlao DS,et al.Specificity of eicosanoid production depends on the TLR-four-stimulated macrophage phenotype.J Leukoc Biol,2011,90:563-574.
15. Lefebvre JS,Marleau S,Milot V,et al.Toll-like receptor ligands induce polymorphonuclear leukocyte migration:key roles for leukotriene B4 and platelet-activating factor. FASEB J,2010,24:637-647.
16. Ruipérez V,Astudillo AM,Balboa MA,et al.Coordinate regulation of TLR-mediated arachidonic acid mobilization in macrophages by group IVA and group V phospholipase A2s.J Immunol,2009,182:3877-3883.
17. Font-Nieves M,Sans-Fons MG,Gorina R,et al.Induction of COX-2 enzyme and down-regulation of COX-1 expression by lipopolysaccharide(LPS)control prostaglandin E2 production in astrocytes.J Biol Chem,2012,287:6454-6468.
18. Gorina R,Font-Nieves M,Márquez-Kisinousky L,et al.Astrocyte TLR4 activation induces a proinflammatory environment through the interplay between MyD88-dependent NF-κB signaling,MAPK,and Jak1/Stat1 pathways.Glia,2011,59:242-255.
19. Lu J,Jilling T,Li D,et al.Polyunsaturated fatty acid supplementation alters proinflammatory gene expression and reduces the incidence of necrotizing enterocolitis in a neonatal rat model.Pediatr Res,2007,61:427-432.
20. Hassan IR,Gronert K. Acute changes in dietary omega-3 and omega-6 polyunsaturated fatty acids have a pronounced impact on survival following ischemic renal injury and formation of renoprotective docosahexaenoic acid-derived protectin D1.J Immunol,2009,182:3223-3232.
21. Ueda T.Fukunaga K.Seki H,et al.Combination Therapy of 15-Epi-Lipoxin A4 With Antibiotics Protects Mice From Escherichia coli-Induced Sepsis.Crit Care,Med 2014.
22. Eisen DP.Manifold beneficial effects of acetyl salicylic acid and nonsteroidal anti-inflammatory drugs on sepsis. Intensive Care Med,2012.
23. Hennekens CH,Schneider WR,Pokov A,et al.A randomized trial of aspirin at clinically relevant doses and nitric oxide formation in humans.J Cardiovasc Pharmacol Ther,2010,15:344-348.
24. Eisen DP,Reid D,McBryde ES.Acetyl salicylic acid usage and mortality in critically ill patients with the systemic inflammatory response syndrome and sepsis.Crit Care Med,2012,40(6):1761-1767.
25. Spite M,Norling LV,Summers L,et al.Resolvin D2 is a potent regulator of leukocytes and controls microbial sepsis. Nature,2009,461:1287-92.
26. Bitto A,Minutoli L,David A,et al.Flavocoxid,a dual inhibitor of COX-2 and 5-LOX of natural origin,attenuates the inflammatory response and protects mice from sepsis. Crit Care,2012,16:R32.
27. Lawrence T,Gilroy DW.Chronic inflammation:a failure of resolution?Int J Exp Pathol,2007,88(2):85-94.
28. Gilroy DW,Lawrence T,Perretti M,et al.Inflammatory resolution:new opportunities for drug discovery.Nat Rev Drug Discov,2004,3(5):401-416.
29. Maldonado-Pérez D,Golightly E,Denison FC,et al.A role for lipoxin A 4as anti-inflammatory and proresolution mediator in human parturition.FASEB J,2011,25(2):569-575.
30. Maderna P,Godson C.Mediators and receptors in the resolution of inflammation.Review,Lipoxins:resolutionary road. British Journal of Pharmacology,2009,158:947-959.
31. Jin S,Zhang L,Lian Q,et al.Post-treatment with aspirintriggered lipoxin A4 analog attenuates lipopolysaccharide induced acute lung injury in mice.the role of hemeoxygenase-1.Anesth Analg,2007,104:369-77.
32. Walker J,Dichter E,Lacorte G,et al.Lipoxin A4 increases survival by decreasing systemic inflammation and bacterial load in sepsis.Shock,2011,36:410-416.
33. Norling LV,Spite M,Yang R,et al.Cutting edge:humanized nano-proresolving medicines mimic inflammation-resolution and enhance wound healing.J Immunol,2011,186:5543-5547.
34. Das UN.Influence of polyunsaturated fatty acids and their metabolites on stem cell biology.Nutrition,2011,27:21-25.
35. Das UN.Essential fatty acids and their metabolites as modulators of stem cell biology with reference to inflammation,cancer,and metastasis. Cancer Metastasis Rev,2011,30:311-324.